Contents page of the issue December 2007




RUMINANTS
M. Keresztes, V. Faigl, A. Márton, Z. Ihnáth, M. Kulcsár, M. Mézes, F. Husvéth, Gy. Huszenicza: Effect of by-pass fat supplementation of the feedstuffs on reproduction in ruminants. Literature review. Part 2 / 707
F. J. Conraths, M. Kramer, C. Freuling, B. Hoffmann, Ch. Staubach, J. Gethmann, J. Teifke, Th. C. Mettenleiter, M. Beer: Bluetongue. Clinical picture, epidemiology, diagnostics – experiences in Germany / 713


PORCINE
T. Süveges, T. Révész: Pathogenesis, detection and control of important respiratory diseases in pigs (summarising article). Part 2. Prevention and treatment / 718

SMALL ANIMALS
Ch. W. Lombard, A. Kovacevic, S. Riesen: What is the direction of cardiology in the small animal practice? / 725

PET ANIMALS
J. Gál, T. Sátorhelyi, G. Andocs, L. Balogh, Z. Szabó: Clinicopathology of some abnormalities of the casque of veiled chameleons (Chamaeleo calyptratus) / 730

TOXICOLOGY
J. Fodor, K. Balogh, M. Weber, M. Mézes, L. Kametler, R. Pósa, V. Rajli, J. Bauer, P. Horn, F. Kovács, M. Kovács: In vivo and in vitro examination of the metabolism of fumonisin B1 / 735

ACADEMIC REPORTS, 2007
Veterinary research in 2006 in the light of academic reports. Part 3 / 746

OPINION
About authorial ethics of our professional articles (F. Karsai) / 758

EVENTS
17th meeting of the Society of Reproductive Biology (4–5th October 2007, Keszthely) (F. Flink) / 760
17th congress of the European College of Veterinary Internal Medicine – Companion Animals (12–15th September 2007, Budapest) (K. Vörös) / 761
 
BOOK REVIEW
A. Jaggy (ed.): Atlas and textbook of neurology of dogs and cats – with CD-ROM (Vörös K.) / 745
T. Kassai: The life and achievement of Sándor Kotlán (J. Mészáros) / 762
Z. Diószegi Z. (ed.): Small animal orthopaedics (L. Tamás) / 763
 
IN MEMORIAM
János Haraszti (1924–2007) (L. Solti) / 765
Márius Padányi Márius (1925–2007) (E. Bezsilla) / 766
Gyula Povazsán (1926–2007) / 767

M. Keresztes – V. Faigl – A. Márton – Z. Ihnáth – M. Kulcsár – M. Mézes – F. Húsvéth – Gy. Huszenicza:
EFFECT OF BY-PASS FAT SUPPLEMENTATION OF THE FEEDSTUFFS ON REPRODUCTION IN RUMINANTS. LITERATURE REVIEW. PART 2

In the second part of the review the possible mechanisms of fat supplementation on reproduction in dairy cattle are discussed. The effect of fat feeding on energy status and on some metabolic hormones is described. Thereafter, the role of fatty acids – mostly some particular polyunsaturated fatty acids – on ovarian steroid hormone and prostaglandin synthesis is detailed.

T. Süveges – T. Révész:
PATHOGENESIS, DETECTION AND CONTROL OF IMPORTANT RESPIRATORY DISEASES IN PIGS (SUMMARISING ARTICLE). PART 2. PREVENTION AND TREATMENT

The authors summarise the knowledge relating to the prevention, and treatment of certain porcine respiratory diseases, especially mycoplasmosis and actinobacillosis.
Beside control based on specific immunoprophylaxis and medical prevention, they analyse the opportunities of non-specific control.
They consider important to prevent the introduction of infectious agents and dissemination in the herd, and for this purpose the compulsory isolation before population and coordinated operation of the production units based on their capacity, all in-all out system, cleansing and disinfection of buildings and equipments and the increase in resistance of pigs by proper feeding and adequate environment factors.
From the control methods based on immunoprophylaxis they present the vaccination against swine influenza, PRRS, actinobacillosis and mycoplasmosis. In case of the two last diseases, they specially deal with the maternal antibodies adversely influencing the value of vaccination.
They summarise in a table the antibiotics and chemotherapeutic products effective for the prevention and treatment of porcine respiratory diseases, emphasising the importance of aimed antibiotic and other medical treatment based on laboratory examinations.
 
J. Gál – T. Sátorhelyi – G. Andocs – L. Balogh – Z. Szabó:
CLINICOPATHOLOGY OF SOME ABNORMALITIES OF THE CASQUE OF VEILED CHAMAELEONS (CHAMAELEO CALYPTRATUS)
 
The authors review diseases found during their diagnostic-curative work causing pathologic deformities of the casque of veiled chamaeleons (Chamaeleo calyptratus).
Veiled chamaeleons raised in captivity are often rachitic, which results in anomalous development and distortion of the bony casque. Additionally, half-acute, chronic inflammations, abscesses may occur in the casque affected by various bacteria, also causing distortion. Very rarely tumors (such as fibrosarcoma diagnosed by the authors) result in pathologic deformities of the casque.

J. Fodor – K. Balogh – M. Weber – M. Mézes – L. Kametler – R. Pósa – V. Rajli – J. Bauer – P. Horn – F. Kovács – M. Kovács:
IN VIVO AND IN VITRO EXAMINATION OF THE METABOLISM OF FUMONISIN B1

The authors investigated the effect of swine ileal and caecal microbiota on fumonisin B1 (FB1) metabolism. In order to determine the absorption and biotransformation of FB1, T-cannula was implanted into the distal part of pigs’ ileum and the total quantity of urine and faeces was collected during the feeding of diet containing toxin (45 mg FB1/kg, for 10 days) and thereafter (for 10 days). At the end of trial several organs, muscle and fat samples were also collected. It could be established that the accumulative absorption of fumonisin B1 was 4%. In the chymus, the FB1 conversion to aminopentol (AP1) and partially hydrolysed FB1 (PHFB1) was 1% and 3.9%, respectively. In the faecal content the main product was the PHFB1 (47%), with small amounts (12%) of AP1. In the examined tissues the efficacy of the FB1 conversion to aminopentol and partially hydrolysed FB1 was 30% and 20%, respectively. In the in vitro study, suspensions of caecal contents, McDougall buffer and pure FB1 (5 µg/ml) were incubated under anaerobic conditions for 0, 12, 24, 48 and 72 h. In the 48th h, the conversion of FB1 into PHFB1 (46%), was nearly equal to the percentile ratio of FB1, while in the 72nd hours it was 49%. In vitro, the conversion of fumonisin B1 to AP1 was less than 1%. Summarized, the intestinal microflora of pigs is able to transform the intact FB1 to a similarly toxic substance (PHFB1) or a more toxic metabolite (AP1).